Gap junction heterogeneity in reentrant ventricular tachycardia

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Gap junction heterogeneity in reentrant ventricular tachycardia.

The cells of all tissues are coupled by gap junctions, with the exception of blood and adult skeletal muscle cells. Gap junctions provide for homoeostasis and integrative function by mediating the exchange of ions and chemical second messenger molecules between adjacent cells. The electrical signaling provided by these specialized intercellular junctions is perhaps best understood in the heart,...

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Bundle Branch Reentrant Ventricular Tachycardia

Bundle branch reentrant (BBR) tachycardia is an uncommon form of ventricular tachycardia (VT) incorporating both bundle branches into the reentry circuit. The arrhythmia is usually seen in patients with an acquired heart disease and significant conduction system impairment, although patients with structurally normal heart have been described. Surface ECG in sinus rhythm (SR) characteristically ...

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Peritricuspid reentrant ventricular tachycardia in Ebstein's anomaly.

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Transmural "Scar-to-Scar" Reentrant Ventricular Tachycardia

We describe a scar-related reentrant ventricular tachycardia circuit with a proximal segment in an endocardial basal septal scar and an exit in a region of slow conduction in a non-overlapping region of epicardial basal lateral scar. The 12-lead EKG demonstrates criteria for a basal lateral epicardial VT, however the same morphology could be produced with a longer stim-latency with pace mapping...

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Development of multiform ventricular tachycardia during atrioventricular nodal reentrant tachycardia.

A woman of 18 presented with a supraventricular tachycardia, subsequently shown to be caused by atrioventricular nodal reentry, which abruptly deteriorated to a multiform ventricular tachycardia. She had not received any antiarrhythmic drugs nor did she have any of the disorders that are usually associated with this atypical ventricular tachycardia.

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ژورنال

عنوان ژورنال: Cardiovascular Research

سال: 2006

ISSN: 0008-6363

DOI: 10.1016/j.cardiores.2006.08.015